Research Papers:
CXCR4/CXCL12/CXCR7 axis is functional in neuroendocrine tumors and signals on mTOR
Metrics: PDF 2685 views | HTML 2940 views | ?
Abstract
Luisa Circelli1,*, Concetta Sciammarella2,*, Elia Guadagno3, Salvatore Tafuto4, Marialaura del Basso de Caro3, Giovanni Botti1, Luciano Pezzullo5, Massimo Aria6, Valeria Ramundo2, Fabiana Tatangelo7, Nunzia Simona Losito7, Caterina Ieranò1, Crescenzo D’Alterio1, Francesco Izzo8, Gennaro Ciliberto9, Annamaria Colao2, Antongiulio Faggiano5, Stefania Scala1
1Molecolar Immunology and Immuneregulation, Istituto Nazionale per lo Studio e la Cura dei Tumori - IRCCS Naples “Fondazione G. Pascale”, Naples, Italy
2Departments of Clinical Medicine and Surgery, “Federico II” University of Naples, Italy
3Advanced Biomedical Sciences, Division of Pathology, “Federico II” University of Naples, Italy
4Abdominal Oncology, Istituto Nazionale per lo Studio e la Cura dei Tumori - IRCCS Naples “Fondazione G. Pascale”, Naples, Italy
5Thyroid and Parathyroid Surgery Unit, Istituto Nazionale per lo Studio e la Cura dei Tumori - IRCCS Naples “Fondazione G. Pascale”, Naples, Italy
6Economics and Statistics, “Federico II” University of Naples, Naples, Italy
7Pathology, Istituto Nazionale per lo Studio e la Cura dei Tumori - IRCCS Naples “Fondazione G. Pascale”, Naples, Italy
8Abdominal Surgery, Istituto Nazionale per lo Studio e la Cura dei Tumori - IRCCS Naples “Fondazione G. Pascale”, Naples, Italy
9Scientific Directorate, Istituto Nazionale per lo Studio e la Cura dei Tumori - IRCCS Naples “Fondazione G. Pascale”, Naples, Italy
*These authors have contributed equally to this work
Correspondence to:
Stefania Scala, e-mail: [email protected]
Antongiulio Faggiano, e-mail: [email protected]
Keywords: chemokine, mTOR, NET, clinical outcome, grading
Received: September 18, 2015 Accepted: January 06, 2016 Published: February 26, 2016
ABSTRACT
Objective: To evaluate the possible crosstalk between C-X-C chemokine receptor 4 (CXCR4)/C-X-C motif chemokine 12 (CXCL12)/C-X-C chemokine receptor 7 (CXCR7) axis with the mammalian target of rapamycin (mTOR) pathway in neuroendocrine tumors (NETs).
Methods: Sixty-one human NETs were included into the study. CXCR4/CXCL12/CXCR7 axis and mTOR pathway were assessed by qRT-PCR and immunohistochemistry (IHC). The effect of mTOR inhibitor, RAD001, was evaluated on CXCR4 pathway through proliferation and p-Erk and p-AKT induction. Results: CXCR4/CXCL12/CXCR7 axis and p-mTOR were found to be active and correlated with grading, Ki67 index and tumor stage. mTOR pathway activation significantly correlated with poor prognosis. In human NET cells, CXCL12 induced mTOR signalling while AMD3100 (CXCR4-antagonist) impaired it. The mTOR-antagonist, RAD001, impaired the CXCL12-dependent induction of CXCR4 downstream effectors. Combination of AMD3100 and RAD001 potentiate cell growth inhibition.
Conclusions: CXCR4/CXCL12/CXCR7 axis is active in NETs and signals on mTOR. CXCR4 might be considered a prognostic factor in NETs. Combined treatment with AMD3100 and RAD001 may provide clinical benefits in NET patients with drug-resistant.
All site content, except where otherwise noted, is licensed under a Creative Commons Attribution 4.0 License.
PII: 7738