Research Papers:
ZIP4 silencing improves bone loss in pancreatic cancer
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Abstract
Qiang Zhang1,2,*, Xiaotian Sun2,3,4,5,*, Jingxuan Yang2,4,5, Hao Ding6, Drake LeBrun2, Kai Ding7, Courtney W. Houchen4, Russell G. Postier5, Catherine G. Ambrose8, Zhaoshen Li3, Xiaohong Bi6, Min Li2,4,5
1Department of Orthopedics, General Hospital of the Jinan Military Command, Jinan, Shandong 250031, China
2The Vivian L. Smith Department of Neurosurgery, the University of Texas Medical School at Houston, Houston, TX 77030, USA
3Department of Gastroenterology, Changhai Hospital, Second Military Medical University, Shanghai 200433, China
4Department of Medicine, the University of Oklahoma Health Sciences Center, Oklahoma City, OK 73104, USA
5Department of Surgery, the University of Oklahoma Health Sciences Center, Oklahoma City, OK 73104, USA
6Department of Nanomedicine and Biomedical Engineering, the University of Texas Medical School at Houston, Houston, TX 77030, USA
7Department of Biostatistics and Epidemiology, College of Public Health, the University of Oklahoma Health Sciences Center, Oklahoma City, OK 73104, USA
8Department of Orthopedic Surgery, the University of Texas Medical School at Houston, Houston, TX 77030, USA
*These authors have contributed equally to this work
Correspondence to:
Xiaohong Bi, e-mail: [email protected]
Zhaoshen Li, e-mail: [email protected]
Min Li, e-mail: [email protected]
Keywords: ZIP4, bone, cachexia, pancreatic cancer, RANK/RANKL signaling
Received: May 10, 2015 Accepted: July 06, 2015 Published: July 20, 2015
ABSTRACT
Metabolic bone disorders are associated with several types of human cancers. Pancreatic cancer patients usually suffer from severe nutrition deficiency, muscle wasting, and loss of bone mass. We have previously found that silencing of a zinc transporter ZIP4 prolongs the survival and reduces the severity of the cachexia in vivo. However, the role of ZIP4 in the pancreatic cancer related bone loss remains unknown. In this study we investigated the effect of ZIP4 knockdown on the bone structure, composition and mechanical properties of femurs in an orthotopic xenograft mouse model. Our data showed that silencing of ZIP4 resulted in increased bone tissue mineral density, decreased bone crystallinity and restoration of bone strength through the RANK/RANKL pathway. The results further support the impact of ZIP4 on the progression of pancreatic cancer, and suggest its potential significance as a therapeutic target for treating patients with such devastating disease and cancer related disorders.
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