Oncotarget

Retraction:

Retraction: PDIA6 promotes the proliferation of HeLa cells through activating the Wnt/β-catenin signaling pathway

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Oncotarget. 2024; 15:219-219. https://doi.org/10.18632/oncotarget.28522

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Huijun Gao1,*, Bing Sun2,*, Hailu Fu1, Xinming Chi1, Faming Wang1, Xiaoyu Qi1, Jun Hu1 and Shujuan Shao3

1Department of Histology and Embryology, Dalian Medical University, Dalian, China
2Department of Thoracic Surgery, The First Hospital of Dalian Medical University, Dalian, China
3Key Laboratory of Proteomics, Dalian Medical University, Dalian, China
*These authors have contributed equally to this work

Published: March 14, 2024

This article has been retracted: Two instances of internal image duplication were discovered in this paper. Specifically, the β-actin blots in Figure 4A are duplicates of the β-actin blots in Figure 3A. The PDIA6 panel of Figure 5A is also a duplicate of the PDIA6 panel in Figure 2G. The authors were unable to make corrections using the original data. Therefore, in accordance with the decision of the Science and Technology Department of Dalian Medical University, and with the agreement of all authors, Oncotarget has decided to retract this paper.

Original article: Oncotarget. 2016; 7:53289–53298. DOI: https://doi.org/10.18632/oncotarget.10795

Huijun Gao1,*, Bing Sun2,*, Hailu Fu1, Xinming Chi1, Faming Wang1, Xiaoyu Qi1, Jun Hu1 and Shujuan Shao3

1 Department of Histology and Embryology, Dalian Medical University, Dalian, China

2 Department of Thoracic Surgery, The First Hospital of Dalian Medical University, Dalian, China

3 Key Laboratory of Proteomics, Dalian Medical University, Dalian, China

* These authors have contributed equally to this work

Correspondence to:

Jun Hu, email: [email protected]
Shujuan Shao, email: [email protected]

Keywords:

Received: RECEIVED_DATE     Accepted: ACCEPTED_DATE     Published: March 14, 2024

Copyright: © 2024 Gao et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.