Correction: Loss of  ZNF32  augments the regeneration of nervous lateral line system through negative regulation of  SOX2  transcription

Yuyan Wei; Kai Li; Shaohua Yao; Junping Gao; Jun Li; Yanna Shang; Jie Zhang; Le Zhang; Yanyan Li; Xianming Mo; Wentong Meng; Rong Xiang; Jiankun Hu; Ping Lin; Yuquan Wei

doi:10.18632/oncotarget.27337

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Corrections:

Correction: Loss of ZNF32 augments the regeneration of nervous lateral line system through negative regulation of SOX2 transcription

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Oncotarget. 2019; 10:7179-7180. https://doi.org/10.18632/oncotarget.27337

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Yuyan Wei¹, Kai Li¹, Shaohua Yao², Junping Gao¹, Jun Li¹, Yanna Shang³, Jie Zhang¹, Le Zhang¹, Yanyan Li¹, Xianming Mo³, Wentong Meng³, Rong Xiang⁵, Jiankun Hu⁴, Ping Lin¹, Yuquan Wei²

¹ Division of Experimental Oncology, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, and Collaborative Innovation Center for Biotherapy, Chengdu, P.R. China
² Division of Cancer Biotherapy, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, and Collaborative Innovation Center for Biotherapy, Chengdu, P.R. China
³ Laboratory of Stem Cell Biology, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, and Collaborative Innovation Center for Biotherapy, Chengdu, P.R. China
⁴ Department of Gastrointestinal Surgery and Laboratory of Gastric Cancer, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, and Collaborative Innovation Center for Biotherapy, Chengdu, P.R. China
⁵ Department of clinical medicine, School of Medicine, Nankai University and Collaborative Innovation Center for Biotherapy, Tianjin, P.R. China

Published: December 24, 2019

This article has been corrected: In Figure 6F, the images of P-1-169 group from Figure 5D were accidentally duplicated. The corrected Figure 6 is shown below. The authors declare that these corrections do not change the results or conclusions of this paper.

Original article: Oncotarget. 2016; 7:70420–70436. DOI: https://doi.org/10.18632/oncotarget.11895.

Figure 6: Identification of NLSs in ZNF32 and the localization of NLS mutants. (A, B) ZNF32 NLS 1 (Aa 170-185). (A) Schematic representation of recombinant, GFP-tagged ZNF32 mutant proteins. Lines represent the deleted sequences in the proteins. (B) The subcellular localization of ZNF32 and NLS 1 mutant proteins. Recombinant proteins are shown in green (GFP), and cell nuclei are shown in blue (DAPI). (C, D) ZNF32 NLS 2 (Aa 186-199). The basic amino acids Lys and Arg were replaced with Ala in NLS 2 mutant. (C) Schematic representation of the recombinant, GFP-tagged ZNF32 mutants. Lines represent the deleted sequences in the proteins. (D) The subcellular localization of ZNF32 and NLS 2 mutant. Recombinant proteins are shown in green (GFP), and cell nuclei are shown in blue (DAPI). (E, F) ZNF32 NLS 3 (Aa 227-239). (E) Schematic representation of the recombinant, GFP-tagged ZNF32 mutants. Lines represent the deleted sequences in the proteins. (F) The subcellular localization of ZNF32 and NLS 3 mutant. Recombinant proteins are shown in green (GFP), and cell nuclei are shown in blue (DAPI). Scale bar = 50 μm.

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Corrections:

Correction: Loss of ZNF32 augments the regeneration of nervous lateral line system through negative regulation of SOX2 transcription