Research Papers:
Polymorphisms in mitotic checkpoint-related genes can influence survival outcomes of early-stage non-small cell lung cancer
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Abstract
Hyo Gyoung Kang1,2,*, Seung Soo Yoo3,*, Jin Eun Choi1,2, Mi Jeong Hong1,2, Sook Kyung Do1,4, Cheng Cheng Jin1, Soyoun Kim1, Won Kee Lee5, Sun Ha Choi3, So Yeon Lee3, Hyun Jung Kim3, Shin Yup Lee3, Jaehee Lee3, Seung Ick Cha3, Chang Ho Kim3, Yangki Seok6, Eungbae Lee6, Sukki Cho7, Sanghoon Jheon7 and Jae Yong Park1,2,3,4
1Department of Biochemistry and Cell Biology, School of Medicine, Kyungpook National University, Daegu, Republic of Korea
2Department of Cell and Matrix Research Institute, School of Medicine, Kyungpook National University, Daegu, Republic of Korea
3Department of Internal Medicine, School of Medicine, Kyungpook National University, Daegu, Republic of Korea
4BK21 Plus KNU Biomedical Convergence Program, Department of Biomedical Science, Kyungpook National University, Daegu, Republic of Korea
5Department of Preventive Medicine, School of Medicine, Kyungpook National University, Daegu, Republic of Korea
6Department of Thoracic Surgery, School of Medicine, Kyungpook National University, Daegu, Republic of Korea
7Department of Thoracic and Cardiovascular Surgery, School of Medicine, Seoul National University, Seoul, Republic of Korea
*These authors have contributed equally to this paper
Correspondence to:
Jae Yong Park, email: [email protected]
Keywords: polymorphisms, mitosis, mitotic checkpoint, survival outcome, lung cancer
Received: December 02, 2016 Accepted: May 22, 2017 Published: June 27, 2017
ABSTRACT
This study was conducted to investigate the association between variants in mitotic checkpoint-related genes and clinical outcomes of non-small cell lung cancer (NSCLC). A total of 766 patients with NSCLC who underwent curative surgery were enrolled. Among the 73 variants evaluated, 4 variants were related with survival outcomes. BUB3 rs7897156C>T was associated with worse overall survival under a recessive model (adjusted hazard ratio = 1.58, 95% confidence interval = 1.07–2.33, P = 0.02). AURKB rs1059476G>A was associated with better overall survival under a recessive model (adjusted hazard ratio = 0.64, 95% confidence interval = 0.41–0.99, P = 0.05). PTTG1 rs1895320T>C and RAD21 rs1374297C>G were associated with worse disease-free survival. In the functional study, relative luciferase activity was higher at the BUB3 rs7897156T allele compared to that at the C allele. Western blot showed that the phosphorylation of AKT and mTOR in the AURKB variant-type (M298) was significantly lower than in the AURKB wild-type (T298). We found that 4 variants of mitotic checkpoint-related genes were associated with survival outcomes in patients with surgically resected NSCLC. Particularly, our results suggest that BUB3 rs7897156C>T and AURKB rs1059476G>A are functional variants.
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