Research Papers:
Polymorphisms in nucleotide excision repair genes and risk of primary prostate cancer in Chinese Han populations
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Abstract
Mengyun Wang1,8,*, Qiaoxin Li1,2,*, Chengyuan Gu3, Yao Zhu3, Yajun Yang4,5, Jiucun Wang4,5, Li Jin4,5, Jing He6, Dingwei Ye3, Qingyi Wei1,7,8
1Cancer Institute, Collaborative Innovation Center for Cancer Medicine, Fudan University Shanghai Cancer Center, Shanghai, China
2Department of Pathology, First Affiliated Hospital, Xinjiang Medical University, Urumqi, China
3Department of Urology, Fudan University Shanghai Cancer Center, Shanghai, China
4Ministry of Education Key Laboratory of Contemporary Anthropology, State Key Laboratory of Genetic Engineering, School of life Sciences, Fudan University, Shanghai, China
5Fudan-Taizhou Institute of Health Sciences, Taizhou, Jiangsu, China
6Department of Hepatobiliary Oncology, State Key Laboratory of Oncology in South China, Sun Yat-Sen University Cancer Center, Guangzhou, China
7Duke Cancer Institute, Duke University Medical Center, Durham, NC, USA
8Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China
*M. Wang and Q. Li contributed equally to this work and should be considered co-first authors
Correspondence to:
Dingwei Ye, email: [email protected]
Qingyi Wei, email: [email protected], [email protected]
Keywords: case-control study, prostate cancer, genetic susceptibility, nucleotide excision repair, polymorphism
Received: October 29, 2015 Accepted: November 21, 2016 Published: December 10, 2016
ABSTRACT
Genetic variants of nucleotide excision repair (NER) genes have been extensively investigated for their roles in the development of prostate cancer (PCa); however, the published results have been inconsistent. In a hospital-based case-control study of 1,004 PCa cases and 1,055 cancer-free controls, we genotyped eight potentially functional single nucleotide polymorphisms (SNPs) of NER genes (i.e., XPC, rs2228001 T>G and rs1870134 G>C; XPD, rs13181 T>G and rs238406 G>T; XPG, rs1047768 T>C, rs751402 C>T, and rs17655 G>C; and XPF, rs2276464 G>C) and assessed their associations with risk of PCa by using logistic regression analysis. Among these eight SNPs investigated, only XPC rs1870134 CG/CC variant genotypes were associated with a decreased risk of prostate cancer under a dominant genetic model (adjusted odds ratio [OR] = 0.77, 95% confidence interval [CI] = 0.64–1.91, P = 0.003). Phenotype-genotype analysis also suggested that the XPC rs1870134 CG/CC variant genotypes were associated with significantly decreased expression levels of XPC mRNA in a mix population of different ethnicities. These findings suggested that XPC SNPs may contribute to risk of PCa in Eastern Chinese men.
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